DKA

DKA made clear: what it is, how it develops, classic signs, and the nursing priorities — fluids, insulin, and potassium — at the bedside.

What is DKA? Diabetic Ketoacidosis

Diabetic Ketoacidosis (DKA) is a life-threatening emergency and a major complication of diabetes. It happens when your body doesn't have enough insulin to allow blood sugar into your cells for use as energy.

  • Who gets it? Most common in Type 1 Diabetes, but it can occur in Type 2 Diabetes during periods of extreme illness or stress. It can also be the first sign of undiagnosed Type 1 diabetes.

DKA is defined by a triad of problems:

  1. Hyperglycaemia (High blood sugar)
  2. Ketosis (Ketones in the blood/urine)
  3. Metabolic Acidosis (Too much acid in the blood)

The Pathophysiology (The “Why”)

Understanding the “why” is key to understanding the treatment. It all starts with a severe lack of insulin.

1) Hyperglycaemia

  • Without insulin, glucose can't get into the cells to be used for energy. It just builds up in the bloodstream.
  • The body thinks it's starving, so the liver makes even more glucose (gluconeogenesis).
  • Result: Blood sugar levels skyrocket (>11 mmol/L or 250 mg/dL). The high sugar concentration pulls water out of cells, causing osmotic diuresis and severe dehydration.

2) Ketosis & Acidosis

  • Since cells are starving for glucose, the body breaks down fat for energy (lipolysis).
  • The byproduct of fat breakdown is ketones (acidic).
  • When ketones build up faster than they are cleared, blood pH drops → metabolic acidosis.
  • Acetone, a ketone, is exhaled → classic fruity / nail polish remover breath.

3) Dehydration & Electrolyte Mayhem

  • Osmotic diuresis causes large losses of water and electrolytes (K⁺, Na⁺, phosphate).

🚨 POTASSIUM IS CRITICAL! Initially, serum K⁺ may look normal or high because acidosis drives it out of cells. Total body K⁺ is low. When insulin therapy starts, K⁺ shifts back into cells → rapid, dangerous fall in serum K⁺ (hypokalaemia).

Causes & Triggers (The “5 S’s”)

  • Sepsis (or any infection) — the most common cause
  • Sickness (stomach bug, flu, MI, stroke)
  • Stress (surgery, major physical or emotional trauma)
  • Skipping Insulin (missed doses, pump failure, incorrect technique)
  • Substance Abuse (especially alcohol)
  • May be the initial presentation of undiagnosed Type 1 Diabetes

Signs & Symptoms

  • Classic “3 P’s”
    • Polyuria — excessive urination
    • Polydipsia — excessive thirst
    • Polyphagia — excessive hunger (often absent due to nausea)
  • Acidosis
    • Kussmaul respirations — deep, rapid, sighing breaths (blowing off CO₂)
    • Fruity/acetone breath
    • Nausea, vomiting, severe abdominal pain
  • Dehydration
    • Hypotension, tachycardia
    • Dry mucous membranes, poor skin turgor
  • Neurological
    • Lethargy, confusion, drowsiness → may progress to coma

Nursing Management & Priorities

Think: Fluids, Insulin, Potassium.

1) Fluid Resuscitation (Priority #1!)

  • Start: Large volumes of 0.9% Normal Saline (isotonic) to restore organ perfusion.
  • When to change: When glucose ~14 mmol/L (≈250 mg/dL), switch to fluid with dextrose (e.g., D5 in 0.45% Saline).
  • Why: Prevent hypoglycaemia while insulin continues to fix the acidosis — you are treating the acidosis, not just the sugar.

2) Insulin Therapy (Stop the Ketones)

  • How: Continuous IV insulin (Fixed Rate Insulin Infusion — FRII).
  • Goal: Stop ketogenesis and gradually lower glucose.
  • Caution: Lower glucose slowly (≈3–4 mmol/L per hour) to avoid cerebral oedema.
  • Critical rule: Do not stop the insulin infusion until acidosis resolves (anion gap closed). Overlap with long-acting subcutaneous insulin for 1–2 hours before stopping IV insulin.

3) Electrolyte Management (Watch Potassium)

  • Monitor: Electrolytes, especially K⁺, every 2–4 hours.
  • Anticipate: Most patients need IV potassium added to fluids as insulin drives K⁺ into cells.
  • Cardiac monitoring: Telemetry for potassium-related arrhythmias (peaked T-waves for high K⁺; flattened T/U waves for low K⁺).

Ongoing Nursing Care & Monitoring

  • Hourly: Capillary glucose, vital signs, neurological status (GCS), urine output (strict I&O).
  • Labs q2–4h: VBG and electrolytes — track pH, bicarbonate, and anion gap for resolution.
  • Education (sick day rules):
    • Never stop insulin, even if you can’t eat.
    • Check blood glucose and ketones frequently.
    • Stay hydrated with sugar-free fluids.
    • Know when to call a clinician or go to hospital.

Educational content for learning support; always follow your local clinical guidelines and protocols.

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